Items where Author or Contributor is "Kropf, P"
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Number of items: 18.
2009
Choi, BS;
Kropf, P;
(2009)
Evaluation of T cell responses in healing and nonhealing leishmaniasis reveals differences in T helper cell polarization ex vivo and in vitro.
Parasite immunology, 31 (4).
pp. 199-209.
ISSN 0141-9838
DOI: https://doi.org/10.1111/j.1365-3024.2009.01094.x
Choi, BS;
Martinez-Falero, IC;
Corset, C;
Munder, M;
Modolell, M;
Müller, I;
Kropf, P;
(2009)
Differential impact of L-arginine deprivation on the activation and effector functions of T cells and macrophages.
Journal of leukocyte biology, 85 (2).
pp. 268-77.
ISSN 0741-5400
DOI: https://doi.org/10.1189/jlb.0508310
Modolell, M;
Choi, BS;
Ryan, RO;
Hancock, M;
Titus, RG;
Abebe, T;
Hailu, A;
Müller, I;
Rogers, ME;
Bangham, CR;
+2 more...
Munder, M;
Kropf, P;
(2009)
Local suppression of T cell responses by arginase-induced L-arginine depletion in nonhealing leishmaniasis.
PLoS neglected tropical diseases, 3 (7).
e480.
ISSN 1935-2727
DOI: https://doi.org/10.1371/journal.pntd.0000480
Munder, M;
Choi, BS;
Rogers, M;
Kropf, P;
(2009)
L-arginine deprivation impairs Leishmania major-specific T-cell responses.
European journal of immunology, 39 (8).
pp. 2161-72.
ISSN 0014-2980
DOI: https://doi.org/10.1002/eji.200839041
Müller, I;
Munder, M;
Kropf, P;
Hänsch, GM;
(2009)
Polymorphonuclear neutrophils and T lymphocytes: strange bedfellows or brothers in arms?
Trends in immunology, 30 (11).
pp. 522-30.
ISSN 1471-4906
DOI: https://doi.org/10.1016/j.it.2009.07.007
Full text not available from this repository.
Oberlies, J;
Watzl, C;
Giese, T;
Luckner, C;
Kropf, P;
Müller, I;
Ho, AD;
Munder, M;
(2009)
Regulation of NK cell function by human granulocyte arginase.
Journal of immunology (Baltimore, Md, 182 (9).
pp. 5259-67.
ISSN 0022-1767
DOI: https://doi.org/10.4049/jimmunol.0803523
Full text not available from this repository.
Rogers, M;
Kropf, P;
Choi, BS;
Dillon, R;
Podinovskaia, M;
Bates, P;
Müller, I;
(2009)
Proteophosophoglycans regurgitated by Leishmania-infected sand flies target the L-arginine metabolism of host macrophages to promote parasite survival.
PLoS pathogens, 5 (8).
e1000555.
ISSN 1553-7366
DOI: https://doi.org/10.1371/journal.ppat.1000555
2008
Müller, I;
Hailu, A;
Choi, BS;
Abebe, T;
Fuentes, JM;
Munder, M;
Modolell, M;
Kropf, P;
(2008)
Age-related alteration of arginase activity impacts on severity of leishmaniasis.
PLoS neglected tropical diseases, 2 (5).
e235.
ISSN 1935-2727
DOI: https://doi.org/10.1371/journal.pntd.0000235
2007
Kropf, P;
Baud, D;
Marshall, SE;
Munder, M;
Mosley, A;
Fuentes, JM;
Bangham, CR;
Taylor, GP;
Herath, S;
Choi, BS;
+4 more...
Soler, G;
Teoh, T;
Modolell, M;
Müller, I;
(2007)
Arginase activity mediates reversible T cell hyporesponsiveness in human pregnancy.
European journal of immunology, 37 (4).
pp. 935-45.
ISSN 0014-2980
DOI: https://doi.org/10.1002/eji.200636542
2006
Munder, M;
Schneider, H;
Luckner, C;
Giese, T;
Langhans, CD;
Fuentes, JM;
Kropf, P;
Mueller, I;
Kolb, A;
Modolell, M;
+1 more...
Ho, AD;
(2006)
Suppression of T-cell functions by human granulocyte arginase.
Blood, 108 (5).
pp. 1627-34.
ISSN 0006-4971
DOI: https://doi.org/10.1182/blood-2006-11-010389
Full text not available from this repository.
2005
Kropf, P;
Fuentes, JM;
Fähnrich, E;
Arpa, L;
Herath, S;
Weber, V;
Soler, G;
Celada, A;
Modolell, M;
Müller, I;
(2005)
Arginase and polyamine synthesis are key factors in the regulation of experimental leishmaniasis in vivo.
FASEB journal, 19 (8).
pp. 1000-2.
ISSN 0892-6638
DOI: https://doi.org/10.1096/fj.04-3416fje
Full text not available from this repository.
2004
Antoniazi, S;
Price, HP;
Kropf, P;
Freudenberg, MA;
Galanos, C;
Smith, DF;
Müller, I;
(2004)
Chemokine gene expression in toll-like receptor-competent and -deficient mice infected with Leishmania major.
Infection and immunity, 72 (9).
pp. 5168-74.
ISSN 0019-9567
DOI: https://doi.org/10.1128/IAI.72.9.5168-5174.2004
Full text not available from this repository.
Kropf, P;
Freudenberg, MA;
Modolell, M;
Price, HP;
Herath, S;
Antoniazi, S;
Galanos, C;
Smith, DF;
Müller, I;
(2004)
Toll-like receptor 4 contributes to efficient control of infection with the protozoan parasite Leishmania major.
Infection and immunity, 72 (4).
pp. 1920-8.
ISSN 0019-9567
DOI: https://doi.org/10.1128/IAI.72.4.1920-1928.2004
Full text not available from this repository.
Kropf, P;
Freudenberg, N;
Kalis, C;
Modolell, M;
Herath, S;
Galanos, C;
Freudenberg, M;
Müller, I;
(2004)
Infection of C57BL/10ScCr and C57BL/10ScNCr mice with Leishmania major reveals a role for Toll-like receptor 4 in the control of parasite replication.
Journal of leukocyte biology, 76 (1).
pp. 48-57.
ISSN 0741-5400
DOI: https://doi.org/10.1189/jlb.1003484
Full text not available from this repository.
2003
Herath, S;
Kropf, P;
Müller, I;
(2003)
Cross-talk between CD8(+) and CD4(+) T cells in experimental cutaneous leishmaniasis: CD8(+) T cells are required for optimal IFN-gamma production by CD4(+) T cells.
Parasite immunology, 25 (11-12).
pp. 559-67.
ISSN 0141-9838
DOI: https://doi.org/10.1111/j.0141-9838.2004.00668.x
Full text not available from this repository.
Kropf, P;
Herath, S;
Klemenz, R;
Müller, I;
(2003)
Signaling through the T1/ST2 molecule is not necessary for Th2 differentiation but is important for the regulation of type 1 responses in nonhealing Leishmania major infection.
Infection and immunity, 71 (4).
pp. 1961-71.
ISSN 0019-9567
DOI: https://doi.org/10.1128/IAI.71.4.1961-1971.2003
Full text not available from this repository.
Kropf, P;
Herath, S;
Weber, V;
Modolell, M;
Müller, I;
(2003)
Factors influencing Leishmania major infection in IL-4-deficient BALB/c mice.
Parasite immunology, 25 (8-9).
pp. 439-47.
ISSN 0141-9838
DOI: https://doi.org/10.1111/j.1365-3024.2003.00655.x
Full text not available from this repository.
2002
Kropf, P;
Herath, S;
Tewari, R;
Syed, N;
Klemenz, R;
Müller, I;
(2002)
Identification of two distinct subpopulations of Leishmania major-specific T helper 2 cells.
Infection and immunity, 70 (10).
pp. 5512-20.
ISSN 0019-9567
DOI: https://doi.org/10.1128/IAI.70.10.5512-5520.2002
Full text not available from this repository.