CD95 is required for the early control of parasite burden in the liver of Leishmania donovani-infected mice.

Alexander, CE; Kaye, PM; Engwerda, CR; (2001) CD95 is required for the early control of parasite burden in the liver of Leishmania donovani-infected mice. European journal of immunology, 31 (4). pp. 1199-1210. ISSN 0014-2980 DOI: https://doi.org/10.1002/1521-4141(200104)31:4<1199::aid-immu1199>3.0.co;2-6

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https://doi.org/10.1002/1521-4141(200104)31:4<1199::aid-immu1199>3.0.co;2-6

Abstract

Item Type Article
Keywords Animal, Antigens, CD95/genetics/*immunology, Apoptosis, CD4-Positive T-Lymphocytes/immunology/metabolism/transplantation, Cells, Cultured, Flow Cytometry, Gene Deletion, Genes, RAG-1/genetics, Genetic Predisposition to Disease, Granuloma/immunology/parasitology, In Situ Nick-End Labeling, Interferon Type II/immunology/metabolism, Leishmania donovani/*growth & development/physiology, Leishmaniasis, Visceral/*immunology/parasitology, Liver/immunology/parasitology/pathology, Membrane Glycoproteins/deficiency/genetics/metabolism, Mice, Mice, Inbred BALB C, Mice, Inbred C57BL, Spleen/immunology, Support, Non-U.S. Gov't, Animal, Antigens, CD95, genetics, immunology, Apoptosis, CD4-Positive T-Lymphocytes, immunology, metabolism, transplantation, Cells, Cultured, Flow Cytometry, Gene Deletion, Genes, RAG-1, genetics, Genetic Predisposition to Disease, Granuloma, immunology, parasitology, In Situ Nick-End Labeling, Interferon Type II, immunology, metabolism, Leishmania donovani, growth & development, physiology, Leishmaniasis, Visceral, immunology, parasitology, Liver, immunology, parasitology, pathology, Membrane Glycoproteins, deficiency, genetics, metabolism, Mice, Mice, Inbred BALB C, Mice, Inbred C57BL, Spleen, immunology, Support, Non-U.S. Gov't
Faculty and Department Faculty of Infectious and Tropical Diseases > Department of Infection Biology > Dept of Immunology and Infection (-2019)
Research Centre Leishmaniasis Group
PubMed ID 11298345
ISI 168090100025

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